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Its efficacy is not based on a single pathway but on the synergistic, multi-target effects of its components
However, AD pathology extends beyond amyloid and tau, encompassing a multifaceted array of pathophysiological mechanisms, including chronic neuroinflammation, oxidative stress, mitochondrial dysfunction, impaired proteostasis (the homeostasis of protein folding, stability, and degradation), calcium dysregulation, excitotoxicity, and breakdown of the blood-brain barrier (BBB) [16, 17]
Regulatory T cells in autoimmunity and cancer: a duplicitous lifestyle
Finally, as much as it is important to shed light on the potential good that glutathione performs as a protective molecule, it is equally important to discover and observe in which ways it is certainly not protective and what elements of a compound-conjugate can trigger its toxicity instead
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