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A 2008 study published in the Journal of Medicinal Chemistry demonstrated metabolic effects in rodent models at 50mg/kg, which led early researchers to extrapolate human-equivalent doses between 50100mg daily

Ambiguous restraints to Arg residues in the HIV-TAT have been excluded given the flexibility of this region

showed that glutathione reductase (GR) activity, was lower in hyperthyroid patients.95 In another investigation it has been observed that oxidative stress occurred in liver tissue of hyperthyroid animals probably due to impaired antioxidant defense mechanisms.96,97 As antithyroid drugsinduced hepatotoxicity is deteriorated in experimental models with defected liver protective mechanisms,41,51 the question arises if GD (the disease which antithyroid drugs are prescribed against) can sensitize individuals to the antithyroid medications' adverse hepatic effects (Fig

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