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These essential prerequisites and defense mechanisms inherently suggest that ferroptosis is a metabolic form of cell death ( There is hope that increasing ferroptosis in TNBC could improve the effective treatment rate

As expected, the inhibition of GPx2 expression by siRNA led to significant growth inhibition in both rat and human breast cancer cell lines [19, 98]

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IGF-1 LR3 combines the best of both approaches: an N-terminal extension of 13 arginine-rich residues for steric IGFBP hindrance, Arg3 substitution to definitively abolish BP3 and BP5 binding, and retention of the 70 IGF-1 residues to maintain IGF-1R recognition