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While it has not been conclusively established whether this dysfunction is a cause or consequence of the pathological processes, growing evidence suggests that reduced proteasomal activity may contribute to the accumulation of toxic protein aggregates and the progression of neurodegeneration

Here, we evaluated whether the inhibition of LDHA by FX11 has anti-inflammatory effects on bFLS stimulated with bTNF-

Of course, it does not only dilute the drug, it maintains its sterility during usage

Another possible contributing mechanism is maladaptive neuroplasticity, in which neuronal pathways adapt and conform pathologically to changes in auditory system nerve signaling