10.3390/antiox12111928 89 PeiyuanH.ZhipingH.ChengjunS.ChunqingW.BingqingL.ImamM
This included increased congenital anomales (Wogelius, 2006), increased heart defects (Kallen, Otterbia, Olausson, 2007), general increase in birth defects (Cole et al, 2007), increased holes in heart chambers (Louik, 2007), elevated major birth defects in children exposed to paroxetine (Berard, 2007), increased anencephaly - missing major portions of the brain (Alwas, 2007), 100% increase in asymptomatic congenital heart defects (Merlob, 2009), increased risk of congentical heart defects and increased risk of hypospadias (Reis and Kallen, 2010), increased holes in the heart, (Bakker, 2010), increased neural tube defects & increased heart ventricle defects (Malm, 2011)
Wada, T
g , Left, percent of cell number from Slc33a1 -knockout KP cells expressing a vector control or SLC33A1 cDNA treated with DMSO or tunicamycin for 4 days under indicated concentrations