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Such impairments closely parallel the mitochondrial respiratory deficits and elevated oxidative markers reported in cortical and limbic regions of depressed individuals, which would impair energy-dependent neuroplastic processes, intracellular signal transduction cascades, and activity-dependent protein synthesis required for synaptic maintenance, dendritic remodelling, and adaptive circuit function [138, 143]

Redox reactions in mammalian spermatogenesis and the potential targets of reactive oxygen species under oxidative stress

Zittermann A, Jungvogel A, Prokop S, et al