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This PK-PD disconnect is compatible with a peptide whose mechanism triggers downstream signalling cascades (VEGFR2 activation, fibroblast migration, angiogenesis) that persist well beyond direct peptide presence

In vitro experiments revealed that treatment with Au/Ag NCs and NIR caused the membranes of multidrug-resistant Acinetobacter baumannii (MDR-AB) to shrink with the formation of numerous pores, resulting in severe deformations of the bacteria

The LAL assay that detects them requires specialized reagents and controlled laboratory conditions

A can self-aggregate, progressively creating oligomers and fibrils, which subsequently impair the function of glial cells and neurons, hence hastening the pathological advancement of AD [31]