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Smoking speeds up collagen breakdown in multiple ways: Direct damage to collagen and elastin leads to early wrinkles and slower wound healing [1]

Thus, the NLRP3 inflammasome serves as a key mechanistic link between the initial toxic insult and the ensuing inflammatory response in APAP hepatotoxicity

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A number of antitumor agents, including vinblastine, cisplatin, doxorubicin, camptothecin and several others have exhibited antitumor activity via the ROS-dependent activation of apoptotic cell death, suggesting the potential use of ROS as an antitumor agent (16)