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Besides, reactive glial cells, members of the NVU, are likely to contribute to the permeability of the BBB observed in cerebral hypoxia through downregulation of paracellular proteins such as Claudin-5 (CLDN5), occludin, and ZO-1 (Obermeier et al., 2013)

The AOD 9604 peptide has been extensively studied for its ability to stimulate lipolysis the breakdown of stored fat and inhibit lipogenesis the formation of new fat cells