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Mitochondrial dysfunction can drive senescence via reactive oxygen species (ROS) accumulation [68, 69], and Tat ( morphine) perturbs mitochondrial function, increasing ROS, protein oxidation, and cytokine release in microglia [60, 70]

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Oxid Med Cell Longev 2017:6501046 Tan Y, Yuan Y, Yang X, Wang Y, Liu L (2024) Diagnostic value of oxidation-reduction potential for male infertility: a systematic review and meta-analysis

Resveratrol alleviates cardiac apoptosis following exposure to fenitrothion by modulating the sirtuin1/c-Jun N-terminal kinases/p53 pathway through pro-oxidant and inflammatory response improvements: In vivo and in silico studies