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GNDs exerted a selective inhibition of NO generation without interfering with the accumulation of pro-inflammatory agents by disrupting the synthesis of inducible NO synthase in activated macrophages through up-regulation of eIF2 phosphorylation levels, without disturbing Fe homeostasis (Zhang et al., 2022)

The issue is not glutathione itself

According to our models, the H248P substitution in the VP1 HI loop alters surface charge and induces conformational changes in the adjacent BC and HI loops (Supplementary Fig

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