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Additionally, in order to explore the mechanism of action being neuronal apoptosis, the research team assessed levels of neuroinflammation and glial activation by detecting levels of IL-1-beta, IL-10, and TNF-alpha in the brain

Peptides of microtubule-associated protein tau (MAPT) and glial fibrillary acidic protein (GFAP) also showed increased levels with worsening pathology (Fig

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