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The specific removal of dysfunctional mitochondria through mitophagy is determined by several mechanisms, including the mitochondrial translocation of Parkin, an E3 ubiquitin ligase (Youle and Narendra, 2011

515 The pro-apoptotic molecule CHOP underscores the connection between ER stress and cell fate in MS, with its expression in various cell types suggesting immune-mediated processes contributing to ER stress-associated damage

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Therefore, increasing expression of SIRT1 following SE seems to be neuroprotective by inducing autophagy and other neuroprotective signaling pathway