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Lan L, Murray TS, Kazmierczak BI, He C

Importantly, lupus T cells exhibit mitochondrial dysfunction, which is characterized by elevated mitochondrial transmembrane potential ( m ) or persistent mitochondrial hyperpolarization (MHP) that mediates diminished activation-induced apoptosis and predisposes T cells to pro-inflammatory death via necrosis (Gergely et al

And did I mention that oxidative stress itself also increases inflammation

We find that the binding site of both compounds is identical to that for a similar molecule that remains naturally attached to the virus 7 , and to the binding site observed for the benzene-sulfonamide derivative in complex with Coxsackievirus B3 8 , identifying the biological role for this binding site