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The most common is based on the inhibition of its degradation in the cytosol, which is followed by its translocation to the nucleus, but it can also be activated by the effect of micromolar amounts of H 2 O 2

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Therefore, to further dissociate the effects of GSH and NRF2 on lipid abundance and assess if NRF2 activation alone mimics the effects of liver-specific Gclc deletion, we treated Gclc WT mice with CDDO-methyl (CDDO-Me), a form of bardoxolone with increased bioavailability

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